After sorting out his thoughts, Xu Yun returned to the bookshelves and started looking again for the books he needed.
Unlike before, the two girls who had mentioned Professor Lu and seemed to be physics majors were nowhere to be seen. He had no idea whether they'd found the Newton materials they wanted.
By now, Xu Yun had realized that his earlier behavior had been pretty strange. Fortunately, the University of Science and Technology of China had a wholesome campus culture. At some other schools, he might have been cut down before he'd even finished speaking.
Xu Yun shook his head, pushed the matter aside, and spent half an hour finding three books and several journal issues.
After taking care of the borrowing paperwork, he returned to his little place.
As mentioned earlier, mammals had nine sodium channel subtypes, NaV1.1 through NaV1.9.
Six of them had been studied in considerable depth. Typical practical applications included certain Class I antiarrhythmic drugs.
Quinidine, lidocaine, and propafenone, for example, all worked by blocking sodium channels.
And if you accidentally got bitten by a spider, venomous snake, scorpion, or succubus, the toxins caused harm by acting on sodium channels.
As for insect sodium channels—
Those were a lot more complicated.
In fact, insects had only one or two genes for voltage-gated sodium channel alpha subunits. But two forms of post-transcriptional modification—alternative splicing and RNA editing—gave insect sodium channels their functional diversity.
On top of that, the insect auxiliary beta subunits TipE and TEH1–4 also played important roles in sodium channel expression and regulation.
So Xu Yun couldn't just slap some proteins together and call it a binding formula. The binding kinetics of the specific receptor were the real key.
It was like those little blue pills: taking two hundred minutes to work and taking