Jujutsu Kaisen: Born a Kamo, My Soul Lives in My Blood
Chapter 19

This is an erroneous chapter, with a group chat number inside

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This is Su. This chapter was posted in error and serves as a replacement for Volume Two, Chapter Eleven.

I don't understand why switching volumes requires me to contact all sorts of people, and they won't even let me delete the text.

On top of that, posting a single chapter to correct the mistake requires a thousand-word self-criticism, and I can't repeat anything or they won't let me post it.

So I can only briefly discuss what I plan to write after Chapter Two.

Simply put, I'll find a plausible way to hand Suguru Geto's corpse over to Kenjaku's faction while maximizing Kamo Xu's hatred for Mahito.

This will lay the groundwork for his complete awakening later on, as well as his total cursed energy approaching Sukuna's without limit.

Ultimately, the protagonist will break with cursed spirits completely, resolve to kill every last one of them, and keep working toward that goal.

Some people complained that my previous novel dragged, so with this one, I can only pick up the pace as much as possible, keeping it enjoyable for readers without relying too heavily on the original plot.

For especially exciting parts, or those closely tied to this story's plot, I'll make slight changes, add a little insight into the characters' minds, and include a few of my own interpretations.

Please forgive the occasional typo. As long as you point it out, I'll definitely go back and correct it.

Interesting comments are also more than welcome. After all, my inspiration is limited, and much of this still depends on everyone pooling their efforts.

Finally, there's the issue of Kamo Xu's Domain Expansion. I've racked my brains over it for ages, but I still haven't come up with a reasonable modification.

His incomplete Domain Expansion has already appeared in Volume Two's Night Parade of a Hundred Demons arc, but I plan to save his complete Domain Expansion for Volume Four's Shibuya Incident arc.

In the meantime, I'd like to gather more reader suggestions about additions or reasonable adjustments to the existing Domain Expansion.

To that end, I'm also planning to start a group chat. Once I finish tomorrow's writing, I'll post the group number in the author's note. [It's already posted in the author's note for Volume Two, Chapter Twelve. No calculations needed; the first digit of each number is the mysterious little digit.]

I hope everyone will join and take an active part in the discussions. Of course, please keep insults and attempts to stir up arguments to a minimum.

I hope we can have a wholesome, friendly group chat.

OK, now comes the word-count padding. Feel free to skip it.

The core functional divisions of the temporal lobe.

Different divisions of the temporal lobe perform specific higher neurological functions:

Auditory processing: areas 41 and 42 of the superior temporal gyrus, together with the transverse temporal gyri, form the primary auditory cortex. They receive and analyze sound signals from the medial geniculate body, allowing people to distinguish pitch, volume, and the direction of sound.

Language comprehension: in the dominant hemisphere, usually the left, the posterior portion of the superior temporal gyrus is the sensory language center, or Wernicke's area. This region is specifically responsible for understanding spoken and written language. Damage to it causes receptive aphasia, in which patients can hear sounds but cannot understand their meaning. In addition, the posterior portion of the middle temporal gyrus in the dominant hemisphere may be involved in naming objects.

Smell and taste: the uncus of the hippocampal gyrus is a center for smell and taste, processing odor information.

Memory formation and storage: the hippocampus, located in the medial temporal lobe, is a core structure for forming and storing new memories. It is especially crucial for converting short-term memories into long-term memories.

Regulation of emotions and mental activity: the anterior temporal cortex and the medial limbic system, including the hippocampus, are closely associated with regulating emotions, mental activity, and behavioral patterns.

Visual information processing: the optic radiation fibers deep within the temporal lobe process some visual information and are associated with homonymous visual fields.

Temporal lobe dysfunction and related diseases

Damage to different regions of the temporal lobe causes corresponding functional impairments. Common related diseases include:

Temporal lobe epilepsy: this often manifests as psychomotor seizures, with symptoms including clouded consciousness, disordered speech, automatisms such as purposeless chewing or walking, hallucinations, particularly olfactory hallucinations, illusions, and memory deficits.

Memory impairment: damage to hippocampal structures in particular can cause significant impairment of recent and remote memory. Symptoms are generally more pronounced when the damage is bilateral.

Auditory and balance disorders: damage to the auditory cortex of one temporal lobe may cause mild bilateral hearing loss, while severe bilateral damage can cause complete cortical deafness. The superior temporal gyrus is also a vestibular cortical center, and lesions can cause balance disorders and vertigo.

Speech disorders:

Receptive aphasia: caused by damage to the posterior portion of the superior temporal gyrus in the dominant hemisphere, or Wernicke's area.

Anomic aphasia: damage to the transitional region between the posterior superior temporal gyrus of the dominant hemisphere and the supramarginal gyrus of the parietal lobe may cause difficulty naming objects.

Visual field defects: temporal lobe lesions may affect the optic radiation fibers, causing contralateral homonymous superior quadrantanopia.

Other neurological symptoms: extensive temporal lobe lesions, such as tumors or hemorrhages, may increase intracranial pressure and cause transtentorial herniation, resulting in symptoms such as pupil dilation, loss of the pupillary light reflex, and oculomotor nerve palsy.

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